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Preclinical studies found that treating neuronal, nematode, or rodent AD models with NR resulted in induction of a mitochondrial stress response, including UPR mt and mitophagy, reduced proteotoxic stress, and decreased Aβ accumulation 243

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6 Its activity can be measured directly in peripheral blood mononuclear cells 8 and indirectly by measuring plasma uracil concentrations or the dihydrouracil:uracil (UH2:U) ratio